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Anand Swaminathan
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This Hangout On Air is hosted by Anand Swaminathan. The live video broadcast will begin soon.
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CORD iMedEd Track
Fri, November 6, 2015, 2:30 PM
Hangouts On Air - Broadcast for free

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Earlier this week this article was sent out via twitter "Hemostatic resuscitation is neither hemostatic nor resuscitative in trauma hemorrhage" http://bit.ly/1fL9E26. It has sprouted a conversation about hemostatic resuscitation on twitter that's probably better served here with more ability to chat and discuss.
It's a prospective cohort study showing that during hemostatic resuscitation, patients lactate and coagulopathy worsens until bleeding is definitively controlled. I commented that you need a comparison group to some other treatment to know if hemostatic resuscitation should be abandoned as other approaches may be just as bad. This study can't tell us this as it only looks at one approach. Additionally, I commented that hypoperfusion/anaerobic metabolism isn't where lactate comes from citing this Marik article bit.ly/1jCSpk2 on sepsis. Not the same as trauma (by any stretch) but is the pathophysiology of lactate generation different?
PubMed journal article Hemostatic resuscitation is neither hemostatic nor resuscitative in trauma hemorrhag was found in Unbound MEDLINE. Download PubMed App to iPhone, iPad, Android smartphone and tablet.
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Is anybody else finding it difficult to interpret Figures 2, 3, & 4 "D"? I find it nearly impossible to interpret the chart in the way that it is presented. Also, I am 100% in agreement with Dr. Swaminathan and Dr. Spegal. Nice paper for hypothesis generation, very catchy title, but data as presented does not allow much else IMO.
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