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Adam Black
13,445 followers -
Gotta Punch Nazis till they Cry.
Gotta Punch Nazis till they Cry.

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Resistance School starts tonight

4 sessions in April, Free online Education in Fighting #theTrumpocalypse .

Graduates of the Harvard Kennedy School of Government, and former Democratic Staffers teach ...

4 nights in April


#ResistanceSchool 


Here’s the session schedule:

Wednesday, April 5, 7:00 - 8:30 p.m . EST: Communicating our values in political advocacy, featuring Tim McCarthy

Signup here! https://www.resistanceschool.com/who-we-are-sign-up/

Session1 Syllabus>>>
https://plus.google.com/+AdamBlack/posts/camexsW2F1m

Wednesday, April 12, 6:30 - 8:00 p.m. EST: Mobilizing and organizing our communities, featuring Sara El-Amine

Thursday, April 20, 7:00 - 8:30 p.m. EST: Structuring and building capacity for action, featuring Marshall Ganz

Thursday April 27, 7:00 - 8:30 p.m. EST: Sustaining the resistance long-term, featuring Michael Blake

Sign up below to register your group and we’ll follow up with you with information on logistics and next steps, including the livestream link, detailed instructions for even the least tech-savvy resistors, and a syllabus of suggested readings.





Resistance School is about community. We’re asking you to convene a group to watch together and work as a community to take action - both locally and nationally. During our sessions, we’ll connect members of the Resistance to one another through a nationwide classroom and real-time conversation.

Speakers will provide interactive breakout sessions to allow your group to practice skills and develop plans that fit your local context. We’ll feed questions from the virtual audience back to the speaker to tailor our syllabus to your needs. Convening live across the country, we’ll build the energy we need to move forward together.

After each session, we encourage your group to spend time as a community: stay and chat, plan your action, grab a bite, and build the relationships we need to take back America.

Have questions or suggestions during the session? Send us an email. Tweet us. We’d love to hear from you.


 #PoliticalReform 

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#CultOfDusty Takes Down Dave Rubin

"serves as a distraction while the Right takes power"

Summarizes Dave Rubin's disastrous Reddit AMA : https://www.reddit.com/r/Classical_Liberals/comments/6ipull/hey_this_is_dave_rubin_lets_talk_about_classical



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Tomorrow, we’ll be announcing the latest planet candidate results from beyond our solar system! Our planet-hunting Kepler mission is capable of finding Earth-sized planets in or near the habitable zone, which is the range of distances from a star where liquid water could pool on the surface of a rocky planet. Watch the announcement live at 11 a.m. EDT: http://go.nasa.gov/2sHlXe7 Have questions during the event? Use #AskKepler
Animated Photo

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Painkillers ~ BrianFallon

And we want love / like it was a drug,
All we wanted was a little relief;
And every heart I held in between
They were painkillers to me,
They were painkillers to me.

#Painkillers ~ #BrianFallon


Feel you move through this crowded room,
And I catch myself like a spell fallen under you,
I've been here before
Watching the wheels go round

Everybody that I've ever known
They just ache all night/ and they wake up alone
Yeah, we wait in the dark
For something to put us to sleep

And, oh, my dear, you never know how
'Til the things you've done /come and run you down

And we want love like it was a drug
All we wanted was a little relief
And every heart I held in between
They were painkillers to me
They were painkillers to me

And could you be to me a love supreme?
'Cause we wait out this life just to hold on to anything
'Cause most of the sparks are just sweet little cherry bombs

Oh my dear don't make a sound
'Till somebody burns you and spits you out

And we want love like it was a drug
All we wanted was a little relief
And every heart I held in between
They were painkillers to me
They were painkillers to me

Use me up (use me up)
Yeah and I'll be enough
Come and use me up (use me up)
Oh don't you love the way I drag you down?
Ain't that enough for you now?

Don't you want love like it was a drug?
Yea don't you want a little relief?
From every heart I held in between
They were painkillers to me
They were painkillers to me

And we want love like it was a drug
Yeah, all we wanted was a little relief
And every heart I held in between
They were painkillers to me
They were painkillers to me
Come get your painkillers from me










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Might be a Huge Breakthrough in Pain Treatment , from post surgery, back pain, Neuropathy and RSS

#MedicalBreakthroughs

Cross-Linked Hyaluronic Acid Injection for Neuropathic Pain:
Case presentation and superficial radial nerve injection technique

By John A. Campa, III, MD

Someone please Tag actual MD or Biochemist on G+ to evaluate This for us.

Claims he can interrupt the cytokine Cascade ( chemical neuroimmuno feedback mechanism that causes acute pain to become chronic ) with an injection of Juvederm / Restayne at the pain site. With zero side effects

This all made sense to me until the end, then I can't tell if it's Pseudoscience using advance handwaivium biochemistry beyond my comprehension, or Hard Science.

You got to read this: He literally uses the phrase "spooky Action at a distance", No Joke , to describe treating Peripheral nerve pain , at the Periphery ( hands/ feet ) caused by Central Nervous System lesions.

This is literally the first time I've seen a Science Paper invoke TV Tropes "lampshading" "Refrigerator Logic". *

Lampshading is when a story deliberately focuses on non intuitive ( or illogical ) plotline to suppress skepticism after the fact. You dont hide it, you make it's weakness a strength.

He recommends it post surgery to prevent pain during healing becoming chronic.

By the end I'm not sure if he's name dropping every Buzzword used in every Neuropathy and chronic pain paper --- or it's cutting edge #Noiception Science . It sounds awesome, easy and within current Technology.

#Neuropathy #TheCureForPain

Injection of Juvederm Treats Neuropathic Pain

https://www.practicalpainmanagement.com/meeting-summary/injection-juvederm-treats-neuropathic-pain


https://www.practicalpainmanagement.com/treatments/interventional/injections/cross-linked-hyaluronic-acid-injection-neuropathic-pain

° Refrigerator logic is story logic that seems to makes sense--- up until the time you get up to get a snack

#ScienceSunday #ScienceOnGooglePlus

( Excerpt )

Postsurgical neuropathic pain is a frequent problem, even if the patient was in the best of hands. As with other types of nerve injury pain, postsurgical neuropathic pain is difficult to treat, usually relying on adjuvant pain medications, such as antidepressants and anticonvulsants, and nerve blocks. I have developed a treatment method that uses commercially available cross-linked hyaluronic acid (Restylane and Juvéderm) that provides prolonged, significant relief without side effects.

The first use of cross-linked hyaluronic acid to treat neuropathic pain was presented at the 2015 Annual Meeting of the *American Academy of Pain Medicine*https://www.practicalpainmanagement.com/meeting-summary/injection-juvederm-treats-neuropathic-pain

 in National Harbor, Maryland.1 

In the 34-month retrospective chart review, 15 neuropathic pain patients (7 women, 8 men) with 22 pain syndromes were studied.

The average age of the patients was 51 years, with a mean pain duration of 66 months. The pretreatment average visual analog scale (VAS) pain score was 7.5 out of 10. After treatment, the VAS was reduced to 1.5 out of 10, and the average duration of relief was 7.7 months.

Since I presented my original work, I have treated an additional 75 patients with similar pain syndromes (ie, postherpetic neuralgia, carpal tunnel and tarsal tunnel syndrome, Bell’s Palsy tinnitus and head pain, etc). Due to the likely mechanisms of action at work, I have designated this method of treatment as Cross-Linked Neural Matrix Antinociception (XL-NMA).2  I present a case report of a patient with persistent neck and hand pain after undergoing cervical spine surgery

How It Works: Mechanisms of Action

Table 1 outlines the proposed mechanisms of action, which are multifactorial. They are ordered with regard to their temporal proximity to the evolving antinociception as noted over time—from the most immediate effects in the first 10 minutes after injection to the enduring and prolonged relief noted at 1 year or more in some cases.



CL-HA acts as a physical protective shield that forms a compartment that blunts the activation of spontaneous activity in the C fibers and Remak bundle afferency, as well as any aberrant nociceptive ephapse

.10 Because of CL-HA’s polyanionic nature, its massive molecule (500 MDA to 100 GDa) may completely depolarize the action potential due to the size of its negative charge and prevent any transmission of the signal. The LMW/HMW mismatch correction results in TNFα-stimulated gene 6 protein modulation of the regional inflammatory response. This stabilizes and restores the immunoneural cross-talk dysregulation at the level of the extracellular neural matrix, essentially blocking what is thought to be causing the chronification of pain.11-14

In essence, after injury or insult to the extracellular neural matrix (ECNM), an initial acute phase of overt clinical inflammation supervenes, with attendant tissue swelling and activation of Aδ and C-fiber nociceptors. However, once this becomes chronic, the tissue inflammation and immuno-neural cross-talk becomes persistent but subclinical. Chronification would occur by virtue of a re-entry, positive feedback loop, thereby sustaining and maintaining the proinflammatory, pronociceptive state, blocking entry into the healing and restoration phase (Table 2). It is self-sustaining due to a LMW/HMW-HA mismatch, likely the result of a CD44/CD168 (RHAMM) gene aberration.



Injection of CL-HA at this point results in loop interruption by correcting the LMW/HMW-HA mismatch, permitting interleukin (IL)-1β and TNFα induction of TSG-6 for inflammatory moderation, by modulation and down-regulation of the interaction between LMW-HA and CD44. This then allows for normal progression to the ECNM anti-inflammatory, antinociceptive phase, as CD44 and RHAMM (CD168) are now able to properly interact with HMW-HA. To understand this mechanism, please refer to Table 2, which illustrates the relevant cytokine cascade and neuroimmunology after ECNM injury.

The foregoing considered, CL-HA may be regarded as a super-mega Dalton form of HA. As such, it augments and sustains many times over the restorative and healing molecular biological normative functions of the body’s HMW-HA, including:

Anti-inflammatory response

Inhibition of scar formation

Formation of functional super-structure aggregates

Immunosuppression

Antiangiogenesis

Increased ability to bind fibrinogen for clotting

Stimulation of peripheral blood monocytes

Production of growth factors and matrix components

Thus, the injection of CL-HA into the ECNM may result in:

Interruption of this loop

Correction of the mismatch

Entry into the healing and restoration phase (Table 2)

Returning the ECNM to homeostasis

Spooky Action at a Distance Confirmed?15

When discussing this case report with peers, I am often asked, “But how does a treatment in the periphery, well distal from the offending lesion in the neck, effect change?”

In this case, the known lesions per CR and CT myelography are identified at the spinal segmental levels, C5-C6 and C6-C7 (C6 and C7 nerve roots, respectively).

These lesions compromise both the nerve roots and the anterior spinal cord, which are thereby intimately part of the known root and cord derivation of the radial nerve (ie, C5, C6, C7, C8, T1). And, of course, they would support the ongoing burning pain experienced over the dorsa of both hands. However, to understand this further, one must consider the concept of deafferentation.16

Deafferentation Pain

Deafferentation pain is simply, “…severe spontaneous pain in body parts distal to the injury despite reduced or no sensitivity to external noxious stimuli to that body part (hypoalgesia or analgesia).”16 It may result from any injury to the nervous system, both central and peripheral, including the brain, spinal cord, and peripheral nerves. The deafferentation is believed to be due to the loss of information from the periphery to the brain. More specifically, there is an interruption in the afferent sensory information passing through the spinothalamic tract to the cortex.

The domain of this tract includes the transmission of pain or nociceptive input centrally to the thalamus. Though the precise mechanism remains poorly understood, this model aptly fits the case at hand (ie, there is incomplete deafferentation of those nerve roots and spinal cord segments subserving the radial nerve).

So, applying this to the patient’s burning pain over the dorsa of the hands, in light of mechanism 3 in Table 1, an injury must occur to initiate the cytokine cascade’s proinflammatory, pronociceptive state (Table 2).

This would be derived from the physical injury to the affected nerve roots and spinal cord segments. However, as the ECNM is a continuous and diffuse neuro-immunological, corporeal entity surrounding all neural structures (ie, it is one throughout), then the affected sensory neurons of the involved C6 and C7 nerve roots and spinal cord segments are in both continuous physical contact and neuro-immunological contact with those overlying the dorsum of both hands.

Hence, the injury at a distance is essentially the result of the proximal ECNM’s spooky action at a distance.15  This causes CD44, CD168 (RHAMM) to detect HAT∆, with inflammatory cytokine release of IL-1β, IL-6 and TNFα, which in due course initiates and sustains C fiber and Aδ nociceptor activation distally (Table 2, #3). With the injury of the ECNM surrounding the SRN established distally, it may now be intervened in-situ successfully with XL-NMA to achieve CL-HA LMW/HMW-HA mismatch correction and ICAM-1 (CD54) inflammatory modulation (Table 2, #3-#5 loop).

Denouement

Nonetheless, it is now truly gratifying to reliably obtain enduring relief of severe and intractable symptomatology with a safe and relatively minimally invasive treatment. The technique is generally simple to perform, with perhaps the most challenging aspect being the identification of the target peripheral sensory nerve, neural network, and matrix to inject. However, with standardization of techniques based on common clinical presentations, this will not be difficult.

Summary

I describe the use and technique of targeted SRN neural matrix antinociception injection of cross-linked hyaluronic acid in the successful treatment of neuropathic pain of the dorsum of both hands that occurred in a 60-year-old injured worker who underwent posterior cervical spine decompression. This technique has resulted in enduring relief and proved to be a safe and effective method in this patient. We recommend its routine use be considered early to manage pain in similar patients.






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Cross-Linked Hyaluronic Acid Injection for Neuropathic Pain:
Case presentation and superficial radial nerve injection technique

By John A. Campa, III, MD

Someone please Tag actual MD or Biochemist on G+ to evaluate This for us.

Claims he can interrupt the cytokine Cascade ( chemical neuroimmuno feedback mechanism that causes acute pain to become chronic ) with an injection of Juvederm / Restayne at the pain site. With zero side effects

This all made sense to me until the end, then I can't tell if it's Pseudoscience using advance handwaivium biochemistry beyond my comprehension, or Hard Science.

You got to read this: He literally uses the phrase "spooky Action at a distance", No Joke , to describe treating Peripheral nerve pain , at the Periphery ( hands/ feet ) caused by Central Nervous System lesions.

This is literally the first time I've seen a Science Paper invoke TV Tropes "lampshading" "Refrigerator Logic". *

Lampshading is when a story deliberately focuses on non intuitive ( or illogical ) plotline to suppress skepticism after the fact. You dont hide it, you make it's weakness a strength.

He recommends it post surgery to prevent pain during healing becoming chronic.

By the end I'm not sure if he's name dropping every Buzzword used in every Neuropathy and chronic pain paper --- or it's cutting edge #Noiception Science . It sounds awesome, easy and within current Technology.

#Neuropathy #TheCureForPain

Injection of Juvederm Treats Neuropathic Pain

https://www.practicalpainmanagement.com/meeting-summary/injection-juvederm-treats-neuropathic-pain


https://www.practicalpainmanagement.com/treatments/interventional/injections/cross-linked-hyaluronic-acid-injection-neuropathic-pain

° Refrigerator logic is story logic that seems to makes sense--- up until the time you get up to get a snack

#ScienceSunday #ScienceOnGooglePlus

( Excerpt )

Postsurgical neuropathic pain is a frequent problem, even if the patient was in the best of hands. As with other types of nerve injury pain, postsurgical neuropathic pain is difficult to treat, usually relying on adjuvant pain medications, such as antidepressants and anticonvulsants, and nerve blocks. I have developed a treatment method that uses commercially available cross-linked hyaluronic acid (Restylane and Juvéderm) that provides prolonged, significant relief without side effects.

The first use of cross-linked hyaluronic acid to treat neuropathic pain was presented at the 2015 Annual Meeting of the *American Academy of Pain Medicine*https://www.practicalpainmanagement.com/meeting-summary/injection-juvederm-treats-neuropathic-pain

 in National Harbor, Maryland.1 

In the 34-month retrospective chart review, 15 neuropathic pain patients (7 women, 8 men) with 22 pain syndromes were studied.

The average age of the patients was 51 years, with a mean pain duration of 66 months. The pretreatment average visual analog scale (VAS) pain score was 7.5 out of 10. After treatment, the VAS was reduced to 1.5 out of 10, and the average duration of relief was 7.7 months.

Since I presented my original work, I have treated an additional 75 patients with similar pain syndromes (ie, postherpetic neuralgia, carpal tunnel and tarsal tunnel syndrome, Bell’s Palsy tinnitus and head pain, etc). Due to the likely mechanisms of action at work, I have designated this method of treatment as Cross-Linked Neural Matrix Antinociception (XL-NMA).2  I present a case report of a patient with persistent neck and hand pain after undergoing cervical spine surgery

How It Works: Mechanisms of Action

Table 1 outlines the proposed mechanisms of action, which are multifactorial. They are ordered with regard to their temporal proximity to the evolving antinociception as noted over time—from the most immediate effects in the first 10 minutes after injection to the enduring and prolonged relief noted at 1 year or more in some cases.



CL-HA acts as a physical protective shield that forms a compartment that blunts the activation of spontaneous activity in the C fibers and Remak bundle afferency, as well as any aberrant nociceptive ephapse

.10 Because of CL-HA’s polyanionic nature, its massive molecule (500 MDA to 100 GDa) may completely depolarize the action potential due to the size of its negative charge and prevent any transmission of the signal. The LMW/HMW mismatch correction results in TNFα-stimulated gene 6 protein modulation of the regional inflammatory response. This stabilizes and restores the immunoneural cross-talk dysregulation at the level of the extracellular neural matrix, essentially blocking what is thought to be causing the chronification of pain.11-14

In essence, after injury or insult to the extracellular neural matrix (ECNM), an initial acute phase of overt clinical inflammation supervenes, with attendant tissue swelling and activation of Aδ and C-fiber nociceptors. However, once this becomes chronic, the tissue inflammation and immuno-neural cross-talk becomes persistent but subclinical. Chronification would occur by virtue of a re-entry, positive feedback loop, thereby sustaining and maintaining the proinflammatory, pronociceptive state, blocking entry into the healing and restoration phase (Table 2). It is self-sustaining due to a LMW/HMW-HA mismatch, likely the result of a CD44/CD168 (RHAMM) gene aberration.



Injection of CL-HA at this point results in loop interruption by correcting the LMW/HMW-HA mismatch, permitting interleukin (IL)-1β and TNFα induction of TSG-6 for inflammatory moderation, by modulation and down-regulation of the interaction between LMW-HA and CD44. This then allows for normal progression to the ECNM anti-inflammatory, antinociceptive phase, as CD44 and RHAMM (CD168) are now able to properly interact with HMW-HA. To understand this mechanism, please refer to Table 2, which illustrates the relevant cytokine cascade and neuroimmunology after ECNM injury.

The foregoing considered, CL-HA may be regarded as a super-mega Dalton form of HA. As such, it augments and sustains many times over the restorative and healing molecular biological normative functions of the body’s HMW-HA, including:

Anti-inflammatory response

Inhibition of scar formation

Formation of functional super-structure aggregates

Immunosuppression

Antiangiogenesis

Increased ability to bind fibrinogen for clotting

Stimulation of peripheral blood monocytes

Production of growth factors and matrix components

Thus, the injection of CL-HA into the ECNM may result in:

Interruption of this loop

Correction of the mismatch

Entry into the healing and restoration phase (Table 2)

Returning the ECNM to homeostasis

Spooky Action at a Distance Confirmed?15

When discussing this case report with peers, I am often asked, “But how does a treatment in the periphery, well distal from the offending lesion in the neck, effect change?”

In this case, the known lesions per CR and CT myelography are identified at the spinal segmental levels, C5-C6 and C6-C7 (C6 and C7 nerve roots, respectively).

These lesions compromise both the nerve roots and the anterior spinal cord, which are thereby intimately part of the known root and cord derivation of the radial nerve (ie, C5, C6, C7, C8, T1). And, of course, they would support the ongoing burning pain experienced over the dorsa of both hands. However, to understand this further, one must consider the concept of deafferentation.16

Deafferentation Pain

Deafferentation pain is simply, “…severe spontaneous pain in body parts distal to the injury despite reduced or no sensitivity to external noxious stimuli to that body part (hypoalgesia or analgesia).”16 It may result from any injury to the nervous system, both central and peripheral, including the brain, spinal cord, and peripheral nerves. The deafferentation is believed to be due to the loss of information from the periphery to the brain. More specifically, there is an interruption in the afferent sensory information passing through the spinothalamic tract to the cortex.

The domain of this tract includes the transmission of pain or nociceptive input centrally to the thalamus. Though the precise mechanism remains poorly understood, this model aptly fits the case at hand (ie, there is incomplete deafferentation of those nerve roots and spinal cord segments subserving the radial nerve).

So, applying this to the patient’s burning pain over the dorsa of the hands, in light of mechanism 3 in Table 1, an injury must occur to initiate the cytokine cascade’s proinflammatory, pronociceptive state (Table 2).

This would be derived from the physical injury to the affected nerve roots and spinal cord segments. However, as the ECNM is a continuous and diffuse neuro-immunological, corporeal entity surrounding all neural structures (ie, it is one throughout), then the affected sensory neurons of the involved C6 and C7 nerve roots and spinal cord segments are in both continuous physical contact and neuro-immunological contact with those overlying the dorsum of both hands.

Hence, the injury at a distance is essentially the result of the proximal ECNM’s spooky action at a distance.15  This causes CD44, CD168 (RHAMM) to detect HAT∆, with inflammatory cytokine release of IL-1β, IL-6 and TNFα, which in due course initiates and sustains C fiber and Aδ nociceptor activation distally (Table 2, #3). With the injury of the ECNM surrounding the SRN established distally, it may now be intervened in-situ successfully with XL-NMA to achieve CL-HA LMW/HMW-HA mismatch correction and ICAM-1 (CD54) inflammatory modulation (Table 2, #3-#5 loop).

Denouement

Nonetheless, it is now truly gratifying to reliably obtain enduring relief of severe and intractable symptomatology with a safe and relatively minimally invasive treatment. The technique is generally simple to perform, with perhaps the most challenging aspect being the identification of the target peripheral sensory nerve, neural network, and matrix to inject. However, with standardization of techniques based on common clinical presentations, this will not be difficult.

Summary

I describe the use and technique of targeted SRN neural matrix antinociception injection of cross-linked hyaluronic acid in the successful treatment of neuropathic pain of the dorsum of both hands that occurred in a 60-year-old injured worker who underwent posterior cervical spine decompression. This technique has resulted in enduring relief and proved to be a safe and effective method in this patient. We recommend its routine use be considered early to manage pain in similar patients.






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Notes on Ontopic Sharing

Q: It's a Philosophy of Mind community, so I can share any Philosophy from my mind ?*

Answer No, "Of" means concerning the nature of. About.

Q: Then, whats on topic?

A: The 500 or So + POM tropes listed linked in the Guidelines , Top links , http://www.consc.net http://www.philpapers.org

Q: And that's it?

A: and any other major topics covered in peer reviewed Philosophers.

Q And that's it?

A: No, you can actually talk about almost anything if it's raised in a Philosophical discussion / using those tropes to analyze something.

Because that is literally doing Philosophy.
( Phenomenology of Wookie Handjobs? When is the Death Star a Moral imperative? Does A working knowledge of Elvish prelude reactionary values? Just no listicles and clickbait. )

Q: ...Someone else could Philosophize in the comments if I bring it up

A: Then let them share it , Mr Game show host ; if you don't intend to follow up or Philosophise yourself.

Q: But I'm really mad about X Topic

A: I don't Care. Not our topic

Q: This Politician said a bad thing

A: FML, No!

Q: It's "What it's like to Be a Bat Trump" with references to Thomas Nagel

A: Goddamit, you checkmated me.

#Nagel

Nagel on Bats ...
https://cutonthebiasworkshop.files.wordpress.com/2011/05/nagel-1974-what-is-it-like-to-be-a-bat.pdf

Today I want to address a related question: Does it even make sense to try to figure out what is happening inside Trump's head? I will frame the discussion with reference to a justly famous work in the philosophy of mind: Thomas Nagel's 1974 essay *What Is It Like To Be A Bat?*
https://cutonthebiasworkshop.files.wordpress.com/2011/05/nagel-1974-what-is-it-like-to-be-a-bat.pdf



Here's my hypothesis: Normal humans are similarly unable to understand or explain what it feels like to be Donald Trump, because in some respects Donald Trump is different from normal humans, just as bats—in virtue of their ability to echolocate—are also different from normal humans."


Michael Dorf: What's It Like to Be Inside Trump's Head
http://www.newsweek.com/michael-dorf-whats-it-be-inside-trumps-head-626327


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'Half the country wants me dead....'
Damn, so close to some self insight, Kelly Ann..

+Kelly-Ann Conway , what does that indicate to you about the quality of your services? 
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